Showing posts with label Idiopathic Environmental Intolerance. Show all posts
Showing posts with label Idiopathic Environmental Intolerance. Show all posts

Wednesday, April 27, 2011

Hawaii Governor Signs MCS Proclamation and Thanks MCS America for it's Contributions to the Health and Wellness of the Hawaii Community!

When I was told that the governor of Hawaii had signed a proclamation declaring May 2011 as Multiple Chemical Sensitivity Awareness Month, I was delighted! As I patiently waited for the proclamation to arrive, I never anticipated this perfectly and beautifully worded document thanking MCS America for its contributions to the health and wellness of the Hawaii community! I am honored that Governor Abercrombie and Lt. Governor Brian Shatz have formally recognized MCS America’s efforts.

Since its inception in 2006, MCS America has worked to gain medical, legal, and social recognition for multiple chemical sensitivity (MCS) as a disorder of organic biological origin induced by toxic environmental insults; provided support and referral services to the individuals with multiple chemical sensitivity (MCS), chronic fatigue syndrome (CFS), fibromyalgia (FM), and other environmental illnesses; and worked toward ensuring that environmental toxicants are identified, reduced, regulated, and enforced through promoting effective legislation. This is only possible because of community involvement.

On behalf of MCS America and our MCS community, I humbly thank Governor Abercrombie and Lt. Governor Schatz for this gracious proclamation. Their recognition of MCS America's outreach efforts to people with Multiple Chemical Sensitivity is greatly appreciated. This proclamation will be used to foster awareness of this health crisis, encourage policy and procedure changes to support people with Multiple Chemical Sensitivity, and make public services and places accessible to people with MCS in Hawaii and all across America.

To view and download the 2011 Hawaii proclamation in PDF format, see http://www.mcs-america.org/hawaii2011mcs.pdf.

A special thank you also goes to Susie Collins at the Canary Report for working with me on the proclamation request. Susie operates one of the best online social networks in the U.S. for people with Multiple Chemical Sensitivity. For more information, visit http://www.thecanaryreport.org/.

Every year countless individuals and organizations across the United States request proclamations for many forms of environmental illness, including:

Multiple Chemical Sensitivity

Electromagnetic Sensitivity

Toxic Injury

Indoor Toxic Mold

Chemical Awareness

Food Allergy

Neuroendocrineimmune Disorders

Chronic Fatigue Syndrome

Fibromyalgia

Myalgic Encephalomyelitis

Gulf War Illness

For more information on how to request a proclamation and plan for awareness events, download a free event planning kit at http://tinyurl.com/69dexwx.

Para obtener más información sobre cómo solicitar una proclama y el plan de actividades de sensibilización, consulte http://tinyurl.com/6ko73c2.


With Aloha,


Lourdes Salvador


Key Words: multiple chemical sensitivity, chemical sensitivity, chemical sensitivities, multiple chemical sensitivities, MCS, EI, environmental illness, sick building syndrome, idiopathic environmental intolerance, fibromyalgia, chronic fatiuge, FM, CFS, mold illness, clinical ecology, alternative medicine, environmental medicine, neuropathy, encephalopathy, toxic, chemical

Saturday, June 5, 2010

CNN: Toxic America, Environmental Dangers



Key Words: multiple chemical sensitivity, chemical sensitivity, chemical sensitivities, multiple chemical sensitivities, MCS, EI, environmental illness, sick building syndrome, idiopathic environmental intolerance, fibromyalgia, chronic fatiuge, FM, CFS, mold illness, clinical ecology, alternative medicine, environmental medicine, neuropathy, encephalopathy, toxic, chemical

CNN: How the US and EU Regulate Chemicals



Key Words: multiple chemical sensitivity, chemical sensitivity, chemical sensitivities, multiple chemical sensitivities, MCS, EI, environmental illness, sick building syndrome, idiopathic environmental intolerance, fibromyalgia, chronic fatiuge, FM, CFS, mold illness, clinical ecology, alternative medicine, environmental medicine, neuropathy, encephalopathy, toxic, chemical

CNN: Living Life Without Plastics

CNN: Living Life Without Plastics


Key Words: multiple chemical sensitivity, chemical sensitivity, chemical sensitivities, multiple chemical sensitivities, MCS, EI, environmental illness, sick building syndrome, idiopathic environmental intolerance, fibromyalgia, chronic fatiuge, FM, CFS, mold illness, clinical ecology, alternative medicine, environmental medicine, neuropathy, encephalopathy, toxic, chemical

CNN: Toxic Chemicals Enter the Womb

CNN: Toxic Chemicals Enter the Womb



Key Words: multiple chemical sensitivity, chemical sensitivity, chemical sensitivities, multiple chemical sensitivities, MCS, EI, environmental illness, sick building syndrome, idiopathic environmental intolerance, fibromyalgia, chronic fatiuge, FM, CFS, mold illness, clinical ecology, alternative medicine, environmental medicine, neuropathy, encephalopathy, toxic, chemical

Thursday, April 1, 2010

Black Mold Exposure Documentary

Black Mold Exposure Documentary
http://thetruthaboutmcs.blogspot.com/2010/04/black-mold-exposure-documentary.html


Key Words: multiple chemical sensitivity, chemical sensitivity, chemical sensitivities, multiple chemical sensitivities, MCS, EI, environmental illness, sick building syndrome, idiopathic environmental intolerance, fibromyalgia, chronic fatiuge, FM, CFS, mold illness, clinical ecology, alternative medicine, environmental medicine, neuropathy, encephalopathy, toxic, chemical

Thursday, December 17, 2009

Chemical sensitivities can cause depression and fatigue, Joan Mathews Larson, PhD

http://thetruthaboutmcs.blogspot.com/2009/12/chemical-sensitivities-can-cause.html



Key Words: multiple chemical sensitivity, chemical sensitivity, chemical sensitivities, multiple chemical sensitivities, MCS, EI, environmental illness, sick building syndrome, idiopathic environmental intolerance, fibromyalgia, chronic fatiuge, FM, CFS, mold illness, clinical ecology, alternative medicine, environmental medicine, neuropathy, encephalopathy, toxic, chemical

Chemical sensitivities can cause depression and fatigue, Joan Mathews Larson, PhD



Key Words: multiple chemical sensitivity, chemical sensitivity, chemical sensitivities, multiple chemical sensitivities, MCS, EI, environmental illness, sick building syndrome, idiopathic environmental intolerance, fibromyalgia, chronic fatiuge, FM, CFS, mold illness, clinical ecology, alternative medicine, environmental medicine, neuropathy, encephalopathy, toxic, chemical

Friday, May 1, 2009

MCS America Celebrates Multiple Chemical Sensitivity and Toxic Injury Awareness Month

In the United States alone, it is estimated that more than over 48 million men, women, and children suffer adverse health reactions to everyday chemicals.

Multiple chemical sensitivity and toxic injury is an increasing epidemic which leads to emergency department visits, job loss, homelessness, school absenteeism, and serious lifelong chronic illness in America. Multiple chemical sensitivity (MCS) affects over 48 million men, women, and children irregardless of gender, race and economic status.

In response to these alarming statistics, and in an effort to raise public awareness of the risks faced everyday by Americans living with multiple chemical sensitivity and toxic injury in May is Multiple Chemical Sensitivity and Toxic Injury Awareness Month in America.

Twenty-five governors and mayors across America have issued proclamations for this important event.

Events are being held across the United States during May. Each event will raise awareness about indoor and outdoor pollutants that trigger multiple chemical sensitivity and toxic injury, as well as ways to prevent toxic injuries and exposures.

MCS is a Major Public Health Concern

At least 45 million people in the US report sensitivity to various chemicals.[1]
About 3 million Americans are diagnosed with MCS.[1]
MCS affects people of all ages, economic status, race, and both genders.[1]
Chemicals that people with MCS react to are toxic and affect everyone to some extent.

Medical Findings

Brain scans show reduced blood flow to the brain when people with MCS are under chemical exposure.[2-7]
Physical variances identified in MCS include brain inflammation, oxidative stress, excitotoxicity, cardiac and airway disease, and auto-immune disorders.[8-14]
Mast cell activation and disorders of porphyrin metabolism have been linked to MCS.[15]
Genetic variations relating to detoxification processes have been linked to MCS.[16]

The Cost of Environmental Illnesses

Annual expenditures for healthcare and lost productivity due to MCS are estimated at $71.8 billion dollars per year.[18]

Environmental Factors

Indoor and outdoor environmental exposures can trigger reactions: perfumes and fragrances, cleaning solutions, scented laundry products, pesticides, herbicides, paint and building materials, gasoline and petroleum based products, artificial flavors, colors, and preservatives.[12]

MCS Can be Controlled

MCS can be controlled with a plan that includes avoidance and control of environmental triggers, many people with MCS can lead normal, healthy, and active lives.[18]

For more information on MCS America’s awareness program, visit www.mcs-america.org.

References

Kreutzer R, Neutra RR, & Lashuay N. Prevalence of people reporting sensitivities to chemicals in a population-based survey. Am J Epidemiol. 1999 Jul 1;150(1):1-12.

Heuser G, Mena I. Neurospect in neurotoxic chemical exposure demonstration of long-term functional abnormalities. Toxicol Ind Health. 1998;Nov-Dec;14(6):813-27.

Callender, TJ, et al. Three-dimensional brain and metabolic imaging in patients with toxic encephalopathy. Environmental Res. 1993;60: 295-319.

Callender, TJ, et al. Evaluation of chronic neurological sequelae after acute pesticide exposure using SPECT brain scans. Journal Toxicology & Environmental Health. 1995;41:275-284.

Heuser, G, et al. Neurospect findings in patients exposed to neurotoxic chemicals. Toxicology & Industrial Health. 1994;10:561-571.

Ross GH, Rea WJ, Johnson AR, Hickey DC, and Simon TR: Neurotoxicity in single photon emission computed tomography brain scans of patients reporting chemical sensitivities. Toxicol Ind Health 1999;15(3-4):415-420.

Simon TR, Hickey DC, Fincher CE, Johnson AR, Ross GH and Rea WJ: Single Photon Emission Computed Tomography of the brain in patients with chemical sensitivities. Toxicol Ind Health 1994;10:573-577.

Elofsson, S, et. a. Exposure to organic solvents. Scandinavian Journal of Work & Environmental Health. 1980;6:239-273.

Seppalainen, AM, et al. Neurophysiological effects of long-term exposure to a mixture of organic solvents. Scandinavian Journal of Work & Environmental Health. 1978;4:304-314.

Jonkman, EJ, et al. Electroencephalographic studies in workers exposed to solvents or pesticides. Electro Clinical Neurophysiology. 1992;82:439-444.

Bokina, AI, et al. Investigation of the mechanism of action of atmospheric pollutants on the central nervous system and comparative evaluation of methods of study. Environmental Health Perspectives. 1976;13:37-42.

Ziem, G. and McTamney, J. Profile of patients with chemical injury and sensitivity. Environ Health Perspect 1997;105:417-436.

Bell I.R. Baldwin, C.M. and Schwartz, G.E. Illness from low levels of environmental chemicals: relevance to chronic fatigue syndrome and fibromyalgia. Am J Med. 1998;105:74S-82S.

Baldwin, CM and Bell, IR. Increased cardiopulmonary disease risk in a community-based sample with chemical odor intolerance: implications for women's health and health- care utilization. Arch Environ Health 1998;53:347-353.

Heuser, G. and Kent, P. 1996. Mast cell disorder after chemical exposure. 124th Annual Meeting of the American Public Health Association, New York NY, 20 November 1996.

Zeim, G, McTamney, J. Profile of patients with chemical injury and sensitivity. Environ Health Perspect. 1997 March; 105(Suppl 2): 417–436.

Fox RA, Joffres MR, Sampalli T, Casey J. The impact of a multidisciplinary, holistic approach to management of patients diagnosed with multiple chemical sensitivity on health care utilization costs: an observational study. J Altern Complement Med. 2007 Mar;13(2):223-9.

Gibson, P. Perceived treatment efficacy for conventional and alternative therapies reported by persons with multiple chemical sensitivity. Environmental Health Perspectives. 2003;111:12,1498–1504.

Key Words: multiple chemical sensitivity, chemical sensitivity, chemical sensitivities, multiple chemical sensitivities, MCS, EI, environmental illness, sick building syndrome, idiopathic environmental intolerance, fibromyalgia, chronic fatiuge, FM, CFS, mold illness, clinical ecology, alternative medicine, environmental medicine, neuropathy, encephalopathy, toxic, chemical

Tuesday, October 28, 2008

NEWSLETTER: MCS America News - November 2008



MCSA NEWS
October 2008, Volume 3, Issue 11

Entire PDF Edition: http://mcs-america.org/november2008.pdf (View, Download, and Print)
NEW! Entire Online Edition:
http://mcs-america.org/mcsanewsnovember2008.htm (View as a Webpage)

Direct Links to Articles Inside This Issue:


Oregon Appellate Court Rules MCS Testimony Admissible

PDF Version: http://mcs-america.org/November2008pg123.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Oregon_Appellate_Court


Fibromyalgia Diagnosis and Treatment - Part 1

PDF Version: http://mcs-america.org/November2008pg456789.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Fibromyalgia_Diagnosis_and


Hiring an MCS Safe Contractor

PDF Version: http://mcs-america.org/November2008pg1011.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Hiring_an_MCS


Study Says MCS is Related to Chemicals, not Psychology

PDF Version: http://mcs-america.org/November2008pg121314.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Study_Says_MCS


Quick Environmental Exposure and Assessment Inventory

PDF Version: http://mcs-america.org/November2008pg1516.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Quick_Environmental_Exposure


Fragrance Impairs Brain Function

PDF Version: http://mcs-america.org/November2008pg17.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Fragrance_Impairs_Brain


Fibromyalgia: Suddenly You're 80 Years Old!

PDF Version: http://mcs-america.org/November2008pg18.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Fibromyalgia


Pharmaceutical Deception

PDF Version: http://mcs-america.org/November2008pg19.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Pharmaceutical_Deception


Diagnosing Contested and Emerging illnesses

PDF Version: http://mcs-america.org/November2008pg20212223.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Diagnosing_Contested_and


Disability and Social Stigma

PDF Version: http://mcs-america.org/November2008pg242526.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Disability_and_Social


Ten Holiday Survival Tips

PDF Version: http://mcs-america.org/November2008pg27.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Ten_Holiday_Survival


Patient Support and Resources

PDF Version: http://mcs-america.org/November2008pg2829303132333435.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Patient_Support


MCS Community News

PDF Version: http://mcs-america.org/November2008pg36.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_MCS_Community_News_


Featured Research Studies

PDF Version: http://mcs-america.org/November2008pg373839.pdf

Online Version: http://mcs-america.org/mcsanewsnovember2008.htm#_Featured_Research_Studies

Multiple Chemical Sensitivities America
http://www.mcs-america.org

admin@mcs-america.org

Copyrighted © 2008 MCS America

__._,_.___

Md. hospitals, care facilities working to cut their use of toxic pesticides

Md. hospitals, care facilities working to cut their use of toxic pesticides

Johns Hopkins Hospital, the University of Maryland Medical Center and nearly a dozen other health care and retirement facilities are working to eliminate toxic pesticides from their pest control efforts, a move that environmental advocates say is the first like it in the country.

The Maryland Pesticide Network launched the effort in 2005 after conducting a survey of pest-control products and practices in state health care facilities statewide.

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Sunday, October 26, 2008

Perinatal dioxin exposure in The Netherlands – a long-term follow-up

Article Abstract

 

 
http://www.inderscience.com/search/index.php?action=record&rec_id=20934&prevQuery=&ps=10&m=or
 


Title: Perinatal dioxin exposure in The Netherlands – a long-term follow-up
 
 
  Author: Marike M. Leijs, Gavin W. Ten Tusscher, Kees Olie, William M.C. Van Aalderen, Tom Vulsma, Matthijs Westra, Johannes Oosting, Janna G. Koppe   Email author(s)
 
 
  Address: Department of Paediatrics and Neonatology, Emma Children's Hospital, Academic Medical Centre, PO Box 22660, 1100DD Amsterdam, The Netherlands; IBED/ESPM, University of Amsterdam, Roeterstraat Amsterdam, The Netherlands. ' Department of Paediatrics and Neonatology, Westfriesgasthuis, Maelsonstraat 3, 1624 NP Hoorn, The Netherlands. ' IBED/ESPM, University of Amsterdam, Roeterstraat Amsterdam, The Netherlands. ' Department of Paediatrics, Emma Children's Hospital, Academic Medical Centre, PO Box 22660, 1100DD Amsterdam, The Netherlands. ' Department of Paediatrics, Emma Children's Hospital, Academic Medical Centre, PO Box 22660, 1100DD Amsterdam, The Netherlands. ' Zaans Medical Centre, Zaandam, Ziekehuis ''De Heel'', PO Box 210, 1500 EE Zaandam, The Netherlands. ' Ecobaby Foundation, Hollandstraat 6, 3634 AT Loenersloot, The Netherlands.' Department of Paediatrics and Neonatology, Emma Children's Hospital, Academic Medical Centre, PO Box 22660, 1100 DD Amsterdam; The Netherlands' Ecobaby Foundation, Hollandstraat 6, 3634 AT Loenersloot, The Netherlands
 
 
  Journal: International Journal of Environment and Health 2008 - Vol. 2, No.3/4  pp. 429 - 438
 
 
  Abstract: In 1987, a cohort study was initiated in the Amsterdam/Zaandam region of The Netherlands to study possible effects of dioxins on development and growth in a population of breastfed children selected after an optimal pregnancy and delivery and birth weight above 2500 grams. Effects on thyroid hormone metabolism, liver, haematology and immunology, and retinol binding protein were found in neonates. Follow-up was performed at the age of 2½ years, 8–12 years and 13–18 years. Neonatally an increase in free thyroxine and thyroid stimulating hormone was found, indicating a change in set point. An enhanced psychomotor maturation was seen at the age of 2½ years. Negative effects on lung function and on brain development (studied with neurophysiological tests of visuo-motoric and cognitive performance) were demonstrated in the age of 8–12 years. In adolescence preliminary results show a delay of almost 1 year in breast development in girls in association with higher (above 40 ng International Toxic Equivalents dioxin) prenatal dioxin exposure and again a negative effect on innate immunity. The abnormalities found indicate an increased vulnerability for more stressful circumstances. New chemicals comparable to dioxins or contaminated with dioxins should not be allowed on the market, or if already there, must be banned.
 
 
  Keywords: perinatal dioxin exposure; follow-up study; prospective cohort study; breast development; thyroid hormone metabolism; brain development; immunotoxicity; lung function; environmental pollution; The Netherlands; breastfeeding; children; child health; liver; haematology; immunology; retinol binding protein.
 
 
  DOI: 10.1504/IJENVH.2008.020934

Designing and building healthy places for children

Article Abstract

 
http://www.inderscience.com/search/index.php?action=record&rec_id=20927&prevQuery=&ps=10&m=or
 


Title: Designing and building healthy places for children
 
 
  Author: Arthur M. Wendel, Andrew L. Dannenberg, Howard Frumkin   Email author(s)
 
 
  Address: Centers for Disease Control and Prevention, National Center for Environmental Health, 4770 Buford Highway, Atlanta, GA 30341, USA; Centers for Disease Control and Prevention, Preventive Medicine Residency, Atlanta, Georgia, USA. ' Centers for Disease Control and Prevention, National Center for Environmental Health, 4770 Buford Highway, Atlanta, GA 30341, USA. ' Centers for Disease Control and Prevention, National Center for Environmental Health, 4770 Buford Highway, Atlanta, GA 30341, USA
 
 
  Journal: International Journal of Environment and Health 2008 - Vol. 2, No.3/4  pp. 338 - 355
 
 
  Abstract: The design and construction of the built environment have broad implications for the health of children. Healthy places should protect children from injury, pollutants and disease, provide children with a place to be physically active, play and experience nature, and promote a sustainable future. Health promotion can occur at all scales of the built environment, including buildings, communities and global infrastructure. The disabled, poor and other disadvantaged groups may benefit from built environment improvements. These improvements require partnerships among urban planners, engineers, architects, developers, public health practitioners and communities.
 
 
  Keywords: built environment; children; public health; child health; health promotion; healthy places.
 
 
  DOI: 10.1504/IJENVH.2008.020927

The PRONET project: pollution reduction options network to enhance implementation of successful transport and indoor environment practices in Europe

Article Abstract

 
http://www.inderscience.com/search/index.php?action=record&rec_id=20923&prevQuery=&ps=10&m=or
 


Title: The PRONET project: pollution reduction options network to enhance implementation of successful transport and indoor environment practices in Europe
 
 
  Author: Peter Jan Van Den Hazel, Joris Van Loenhout, Pauline Slottje, Rob Jongeneel   Email author(s)
 
 
  Address: Hulpverlening Gelderland Midden (Public Health Services Gelderland Midden), PO Box 5364, 6802 EJ Arnhem, The Netherlands. ' Hulpverlening Gelderland Midden (Public Health Services Gelderland Midden), PO Box 5364, 6802 EJ Arnhem, The Netherlands. ' Centre for Environmental Health Research, National Institute for Public Health and Environment (RIVM), PO Box 1, 3720 BA Bilthoven, The Netherlands. ' Centre for Environmental Health Research, National Institute for Public Health and Environment (RIVM), PO Box 1, 3720 BA Bilthoven, The Netherlands
 
 
  Journal: International Journal of Environment and Health 2008 - Vol. 2, No.3/4  pp. 293 - 303
 
 
  Abstract: The aim of the Pollution Reduction Options Network (PRONET) project is to facilitate exchange and evaluation of interventions on environment and health exposure reduction measures on a regional level, and promote implementation of successful initiatives in other regions of Europe. This project will focus on the exchange of useful practices in two key areas in environmental health policy:(1) the reduction of transport-related health hazards (air pollution and noise) and (2) the improvement of the indoor environment by reducing indoor exposure to environmental health hazards. The collection of measures on reducing indoor exposure is done by means of a call for case studies among a large group of stakeholders in Europe and through literature search. The final results of PRONET include: (1) a platform to aid exchange of useful information and practical experience in improving indoor environment and transport and (2) a searchable database with good practice examples in reducing exposure to indoor environment pollutants and transport-related examples. The total of potentially relevant publications found was 636, of which 48% were related to biological agents, 25% to physical and 21% to chemical factors in the indoor environment.
 
 
  Keywords: public health; transport; Europe; indoor environment; environmental health policy; good practice; pollution reduction; air pollution; noise; environmental hazards; health hazards.
 
 
  DOI: 10.1504/IJENVH.2008.020923

Environmental threats to children's health – a global problem

Article Abstract  

 
http://www.inderscience.com/search/index.php?action=record&rec_id=20922&prevQuery=&ps=10&m=or  


Title: Environmental threats to children's health – a global problem
 
 
  Author: Maria Neira, Fiona Gore, Marie-Noel Brune, Tom Hudson, Jenny Pronczuk de Garbino   Email author(s)
 
 
  Address: Department of Public Health and Environment, World Health Organization, Geneva, Switzerland. ' Department of Public Health and Environment, World Health Organization, Geneva, Switzerland. ' Department of Public Health and Environment, World Health Organization, Geneva, Switzerland. ' Department of Public Health and Environment, Boston University, Boston, MA, USA. ' Department of Public Health and Environment, World Health Organization, Geneva, Switzerland
 
 
  Journal: International Journal of Environment and Health 2008 - Vol. 2, No.3/4  pp. 276 - 292
 
 
  Abstract: Today's children are exposed to a wide range of environmental threats, whose consequences on health and development may appear early in life, throughout their youth and even later, in adulthood. Health problems linked to environmental hazards are multiplying and becoming more visible due to a rapidly changing environment, rapid population growth, overcrowding, fast industrialisation and uncontrolled pollution from many anthropogenic and also natural sources, and as a result of the effects of climate change. A World Health Organization report on the global burden of disease estimated that nearly a quarter of the global disease burden is related to environmental causes. These environmental-mediated diseases cause more than three million deaths in children under five every year. Such a large burden is unacceptable. Both industrialised and developing countries should reinforce their capacities to assess the environmental burden of paediatric diseases and characterise their impact on children's health. This will enable all responsible sectors to distinguish the main environmental threats affecting children's health and identify their specific roles in improving children's environments. Implementing these activities and turning efforts into prevention, education, policy-making and other actions will reduce the burden of disease affecting children globally, therefore contributing towards achieving the Millennium Development Goals (MDGs).
 
 
  Keywords: children; child health; environmental impact; disease burden; global; environmental hazards; paediatric diseases; health risks; health hazards.
 
 
  DOI: 10.1504/IJENVH.2008.020922

Saturday, October 25, 2008

Emerging contaminants in car interiors: evaluating the impact of airborne PBDEs and PBDD/Fs.

Environ Sci Technol. 2008 Sep 1;42(17):6431-6. Links

Emerging contaminants in car interiors: evaluating the impact of airborne PBDEs and PBDD/Fs.

http://www.ncbi.nlm.nih.gov/pubmed/18800511?dopt=AbstractPlus

Environmental Chemical Processes Laboratory, Department of Chemistry, University of Crete, GR-71003 Heraklion, Greece.

Air samples from automobile cabins were collected and analyzed for polybrominated diphenyl ethers (PBDEs), polybrominated dibenzofurans (PBDFs), and polybrominated dibenzo-p-dioxins (PBDDs). The concentration of total PBDEs (sigmaPBDE; sum of 19 congeners) varied from 0.4 to 2644 pg m(-3), with a median of 201 pg m(-3), while BDE 47, 99, and 209 collectively accounted for 70 +/- 30% of sigmaPBDE concentrations. Multiple linear regression analysis revealed that sigmaPBDE concentration was significantly influenced by vehicle's age and interior temperature. More specifically, sigmaPBDE decreased over time and increased with the rise of temperature. The daily inhalation intake of PBDEs during commuting was estimated to range from 0.5 to 2909 pg day(-1) (median 221 pg day(-1)) and contributed 29% of the overall daily exposure to PBDEs via inhalation. When excluding BDE 209, a lower contribution was calculated for this source (18%), but this was still comparable with residential exposure (22%). The levels of PBDD/Fs were generally below the limits of detection and only in one case were hepta-BDFs positively detected at a concentration of 61 pg m(-3). This study demonstrates that car interiors, especially when new, contain high levels of airborne PBDEs and represent a potential route of human exposure via inhalation.

PMID: 18800511 [PubMed - indexed for MEDLINE]

Habituation and sensitization to heat and cold pain in women with fibromyalgia and healthy controls.

Pain. 2008 Oct 21. [Epub ahead of print]Click here to read

Habituation and sensitization to heat and cold pain in women with fibromyalgia and healthy controls.

http://www.ncbi.nlm.nih.gov/pubmed/18947923?dopt=AbstractPlus

Department of Psychology, University of New Mexico, MSC03 2220, Albuquerque, NM 87131, USA.

The purpose of this study was to examine differences in habituation to heat and cold pain in women with fibromyalgia (FM; n=33) and in women who were healthy controls (HC; n=44). Quantitative sensory testing (QST) was used to assess pain thresholds during five consecutive trials of ascending heat and descending cold stimulation. Anxiety, depression, fatigue, and pain during the previous week were assessed using self-report measures. The overall hypotheses were that there would be differences between groups in pain thresholds and in the rate of habituation to heat and cold pain stimuli. Multilevel modeling was used to test the hypotheses. There were large overall differences in pain thresholds, with the FM group showing greater sensitivity to heat and cold pain stimuli compared with the HC group. While habituation occurred in both of the groups for heat pain, the HC group had stronger habituation across trials than the FM group. Conversely, while the HC group habituated to cold pain stimuli, the FM group showed sensitization and had decreased cold pain thresholds across trials (they felt cold pain at higher temperatures). In addition, anxiety, depression, fatigue, and pain were related to decreased heat and cold pain thresholds in the overall sample. However, when group was controlled, none of these variables were related to thresholds or rates of habituation or sensitization. The differences between women with FM and healthy women in habituation and sensitization may have important implications for the etiology, diagnosis, and treatment of FM and other chronic pain conditions.

PMID: 18947923 [PubMed - as supplied by publisher]

Clinical Correlate of Brain SPECT Perfusion Abnormalities in Fibromyalgia.

J Nucl Med. 2008 Oct 16. [Epub ahead of print] Links

Clinical Correlate of Brain SPECT Perfusion Abnormalities in Fibromyalgia.

http://www.ncbi.nlm.nih.gov/sites/entrez/18927329

Service Central de Biophysique et de Médecine Nucléaire, AP-HM Timone, Marseille, France.

The purpose of this study was to investigate the specific clinical correlate of brain SPECT perfusion abnormalities reported in fibromyalgia. METHODS: We performed a whole-brain voxel-based correlation analysis involving regional cerebral blood flow and various parameters related to pain (Visual Analog Scale, Tubingen Pain Behavior Scale, and Questionnaire Douleur de Saint-Antoine Scale), disability (Fibromyalgia Impact Questionnaire [FIQ]), and anxiety and depression status (Hospital Anxiety and Depression scale) in 20 patients with fibromyalgia (P voxel < 0.005). Ten healthy control women were also included, in order to determine areas of significant hypo- and hyperperfusions in patients. RESULTS: FIQ total score was positively correlated with bilateral parietal perfusion, including postcentral cortex. These clusters of correlation were included in the areas of significant hyperperfusion. FIQ total score was also negatively correlated with perfusion of a left anterior temporal cluster, included in the areas of significant hypoperfusions. No other clinical correlation was observed with regional cerebral blood flow. CONCLUSION: These results show that brain perfusion abnormalities in patients with fibromyalgia are correlated with the clinical severity of the disease.

PMID: 18927329 [PubMed - as supplied by publisher]

Thursday, October 23, 2008

Benefits of Reducing Prenatal Exposure to Coal-Burning Pollutants to Children's Neurodevelopment in China.

Environ Health Perspect. 2008 Oct;116(10):1396-400. Epub 2008 Jul 14.

Benefits of Reducing Prenatal Exposure to Coal-Burning Pollutants to Children's Neurodevelopment in China.

http://www.ncbi.nlm.nih.gov/pubmed/18941584?dopt=AbstractPlus

Department of Environmental Health Sciences, Columbia University, New York, New York, USA.

BACKGROUND: Coal burning provides 70% of the energy for China's industry and power, but releases large quantities of polycyclic aromatic hydrocarbons (PAHs) and other pollutants. PAHs are reproductive and developmental toxicants, mutagens, and carcinogens. OBJECTIVE: We evaluated the benefit to neurobehavioral development from the closure of a coal-fired power plant that was the major local source of ambient PAHs. METHODS: The research was conducted in Tongliang, Chongqing, China, where a coal-fired power plant operated seasonally before it was shut down in May 2004. Two identical prospective cohort studies enrolled nonsmoking women and their newborns in 2002 (before shutdown) and 2005 (after shutdown). Prenatal PAH exposure was measured by PAH-DNA adducts (benzo[a]pyrene-DNA) in umbilical cord blood. Child development was assessed by the Gesell Developmental Schedules at 2 years of age. Prenatal exposure to other neurotoxicants and potential confounders (including lead, mercury, and environmental tobacco smoke) was measured. We compared the cohorts regarding the association between PAH-DNA adduct levels and neurodevelopmental outcomes. RESULTS: Significant associations previously seen in 2002 between elevated adducts and decreased motor area developmental quotient (DQ) (p = 0.043) and average DQ (p = 0.047) were not observed in the 2005 cohort (p = 0.546 and p = 0.146). However, the direction of the relationship did not change. CONCLUSION: The findings indicate that neurobehavioral development in Tongliang children benefited by elimination of PAH exposure from the coal-burning plant, consistent with the significant reduction in PAH-DNA adducts in cord blood of children in the 2005 cohort. The results have implications for children's environmental health in China and elsewhere.

PMID: 18941584 [PubMed - in process]

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