Wednesday, March 7, 2012

Mercury Poisoning Linked to Skin Products

Mercury Poisoning Linked to Skin Products
http://www.fda.gov/ForConsumers/ConsumerUpdates/ucm294849

"Federal health officials are warning consumers not to use skin creams, beauty and antiseptic soaps, or lotions that might contain mercury.

The products are marketed as skin lighteners and anti-aging treatments that remove age spots, freckles, blemishes and wrinkles, says Gary Coody, national health fraud coordinator in the Food and Drug Administration's Office of Regulatory Affairs. Adolescents also may use these products as acne treatments, adds Coody. Products with this toxic metal have been found in at least seven states."

Tuesday, March 6, 2012

Draft state school environmental health guidelines (110pp, 1.78 MB, About PDF) are currently available for a 45-day public comment period.

Draft state school environmental health guidelines (110pp, 1.78 MB, About PDF) are currently available for a 45-day public comment period.
http://www.epa.gov/schools/ehguidelines/index.html

Fresh Fruit and Vegetable Program: A Proposed Rule by the Food and Nutrition Service on 02/24/2012

Fresh Fruit and Vegetable Program:  A Proposed Rule by the Food and Nutrition Service on 02/24/2012
https://www.federalregister.gov/articles/2012/02/24/2012-4181/fresh-fruit-and-vegetable-program#p-3

Lawsuit links illnesses to north St. Louis County creek


Water had been tainted by nuclear waste buried in North County.

"FLORISSANT • The children of Coldwater Creek recall carefree days spent on its banks, digging for arrowheads, catching crawfish and collecting cans in the water that snaked through their north St. Louis County neighborhoods.
They say those memories have become clouded as they learn more about nuclear waste sites near the airport that contaminated the creek starting in the 1940s. And they want to know if their cancers, autoimmune disorders, and health issues affecting their children could be linked to time spent living near and playing in the creek."

Diesel exhaust raises risk of dying from lung cancer, study says

Diesel exhaust raises risk of dying from lung cancer, study says
http://www.latimes.com/health/boostershots/la-heb-diesel-exhaust-cancer-20120302,0,2220205.story

"Diesel engine exhaust has long been officially classified as a probable carcinogen. A new study, however, provides evidence that long-term exposure to even low levels of diesel exhaust raises the risk of dying from lung cancer."

Bill to ban 2 potential carcinogens from kids' products dies in House

Bill to ban 2 potential carcinogens from kids' products dies in House
http://www.thenewstribune.com/2012/03/03/2051421/toxic-kids-products-bill-dies.html

"A bill to ban a pair of potentially carcinogenic flame-retardant chemicals from children's products has died in the Washington state House amid budget intrigue in the Senate."

Is aluminium really a silent killer?

Is aluminium really a silent killer?
http://www.telegraph.co.uk/health/9119528/Is-aluminium-really-a-silent-killer.html

"Fresh concerns following the 1988 Camelford water pollution disaster will be aired this week.

Twenty-four years ago, one of the UK's most notorious pollution disasters occurred. At a water treatment works on the edge of Bodmin Moor, 20 tonnes of aluminium sulphate leaked into the water supply serving the nearby town of Camelford.

Years of bitter disputes followed, with people who had drunk the water complaining of health problems. There were government inquiries, accusations of a cover-up – and, in 2004, the death of Carole Cross. This 58-year-old Camelford resident died from a rare and aggressive form of Alzheimer's, and her brain was found to contain unusually high levels of aluminium."

Effects of Environmental Toxicants Reach Down Through Generations

Effects of Environmental Toxicants Reach Down Through Generations
http://www.sciencedaily.com/releases/2012/03/120302101821.htm
"A Washington State University researcher has demonstrated that a variety of environmental toxicants can have negative effects on not just an exposed animal but the next three generations of its offspring.

The animal's DNA sequence remains unchanged, but the compounds change the way genes turn on and off -- the epigenetic effect studied at length by WSU molecular biologist Michael Skinner and expanded on in the current issue of the online journal PLoS ONE."

Coke, Pepsi have carcinogens: CSPI

Coke, Pepsi have carcinogens: CSPI
http://www.stonehearthnewsletters.com/coke-pepsi-have-carcinogens-cspi/food-safety/

"New chemical analyses have found that Coca-Cola, Pepsi-Cola, Diet Coke, and Diet Pepsi contain high levels of 4-methylimidazole (4-MI), a known animal carcinogen. The carcinogen forms when ammonia or ammonia and sulfites are used to manufacture the "caramel coloring" that gives those sodas their distinctive brown colors, according to the Center for Science in the Public Interest, the nonprofit watchdog group that commissioned the tests. CSPI first petitioned the FDA to ban ammonia-sulfite caramel coloring in February 2011."

The doctor diagnosed chronic Lyme disease, but many experts say it doesn't exist

The doctor diagnosed chronic Lyme disease, but many experts say it doesn't exist
http://www.washingtonpost.com/national/health-science/the-doctor-diagnosed-chronic-lyme-disease-but-many-experts-say-it-doesnt-exist/2012/02/06/gIQA4aMHtR_story.html

"I don't believe in the Loch Ness monster. I don't think government scientists are autopsying aliens in Area 51 or plotting a vast conspiracy from a bunker at the North Pole. But for the past two years, I've lived through an experience that has felt, at times, like a real-life episode of "The X-Files."

What happened is this: My husband became frighteningly, mysteriously ill. And when two doctors finally fit the puzzle pieces of his symptoms together, we discovered that, according to medical authorities, his disease does not officially exist."

Setting Things Straight on Electroconvulsive Therapy (ECT)

Setting Things Straight on Electroconvulsive Therapy (ECT)

http://thetruthaboutmcs.blogspot.com/2012/03/setting-things-straight-on.html


Electroconvulsive therapy (ECT) was originally known as electroshock therapy and was widely used to treat depression before the development of psycho pharmaceutical drugs such as antidepressants and anxiolytics.


Electroshock therapy originated in Berlin in 1927 when Manfred J. Sakel accidentally overdosed a diabetic patient with insulin, leading to convulsions and a sudden improvement in the patient's schizophrenia. Sakel proceeded to treat schizophrenics with what came to be known as "Sakel's Technique". Eventually, it was realized that insulin therapy was dangerous, oftentimes resulted in the death of the patient, was temporary, and thus was not a real cure.


In 1934, Ladislaus J. von Meduna developed another method of inducing convulsions with a drug called Metrazol. The convulsions induced by this drug were so dramatic that they resulted in spinal fractures in nearly half the patients. The treatment was considered far too dangerous and was stressful for psychiatrists to perform while the patient screamed and convulsed wildly on the table.


Lobotomies became popular in 1935. A method called the "ice-pick lobotomy" was popularly used on over a million people. The lobotomy involved inserting an ice pick under the eye and moving it around to damage and chop up the pre-frontal cortex of the brain and then retracting it. This left patients a permanently brain damaged zombie state, a benefit for psychiatric hospital staff. But, it was not a cure either.


Electroconvulsive therapy came on the scene in 1937 and was developed by Ugo Cerletti and Lucio Bini after they discovered that shocking animals temporarily stunned them without killing them. They soon turned to administering electroshock therapy to human subjects. ECT was marketed as a more acceptable form of electroshock therapy because patients were restrained and sedated with anesthesia and paralyzing drugs to control their screams and movement. This "more pleasant" way of damaging and numbing the brain with electroshock was used to mislead people to believe it was a new, pleasant, and safe method of effective psychosurgery.


However, many report that ECT is a barbaric process of inducing seizures and convulsions. A few people who have undergone ECT later committed suicide as a result. Others have lived to post their horrific tales of hurt in videos on YouTube. Many have permanent brain damage. Some have died.


By now, readers may be wondering what this has to do with multiple chemical sensitivity (MCS). A new study published in the Journal of Electroconvulsive Therapy (ECT) by Jesper Elberlinig MD, Nils Gulmann, and Alice Rasmussen has declared that "a substantial, positive effect on symptom severity and social disability related to MCS was obtained by an initial ECT course and maintenance treatment. Electroconvulsive therapy should be considered an option in severe and socially disabling MCS."


The researchers of this study claim that "no effective treatment has been reported" for MCS. This is clearly untrue, which leads one to wonder what the ulterior motives of the researchers are. Avoidance and safe housing have both been reported as effective treatments with a statistically significant success ratio of over 95% improved. Everything industry does to discredit people with MCS completely ignores that safe environments alleviate symptoms. It is simply not true that "nothing works".


The study performed ECT on a single subject with MCS. This is not scientifically valid in terms of findings. Those familiar with science and statistics will be familiar with the bell curve. If you take one hundred people and plot their test values on a line, you will usually discover a bell curve. The bell curve has most of the subjects at its highest point, roughly 90 – 95%. A few subjects will fall on either side of the bell and are known as outliers. With a study on a single subject, there is no valid way to determine whether the subject was an average person at the top of the bell, or an outlier.


The results were described in terms of the perceived improvement of "symptom severity and social disability". This is quite different from measured improvements such as reduced hepatic toxicity and better liver function. The abstract does not even provide the diagnostic criteria used to determine the diagnosis; therefore, it can not be determined whether the subject truly had MCS.


In the study, the patient had to undergo ECT eight times and then have bi-weekly ECT treatments to maintain the "improvement". We must consider that more and more people are being injured and developing MCS. If people can't live without going in for bi-weekly ECT, something is wrong with the larger picture of toxic environmental exposures. In nature, we are supposed to be able to survive without being hooked up to machines and drugs. In nature, we DID and DO survive without being hooked up to these things. A safe, clean environment alleviates symptoms and PREVENTS the injuries leading to MCS in the first place.


In another study, Resertson and Pryor emphasize the important truth that "clinicians should fully inform patients of the possible permanent adverse effects of the treatment, which include amnesia, memory disability, and cognitive disability." (Robertson, H. & Pryor, R. (2006) Memory and cognitive effects of ECT: informing and assessing patients. Advances in Psychiatric Treatment, 12,228-237.)


A journal such as this study was published in, which is dedicated to ECT, isn't going to do anything other than find and support ways to use ECT for everything and anything. Even psychiatry has largely abandoned it in favor of psychiatric drugs. If psychiatric drugs don't cure MCS, why would ECT?


The Citizens Commission on Human Rights has some eye opening videos of the true history of psychiatry with their own inadvertent capability of numbing one from the horrors of what psychiatry is really about. Be patient while the site lead and then go to the "museum" on the top menu. This video covers the history of ECT specifically.


No matter how you look at it, ECT is not a cure and certainly not an ideal thing to keep doing to the sensitive brain, particularly if one is electrosensitive.

Monday, March 5, 2012

Effect of an Integrative Medicine Course on Knowledge Base and Practice Patterns (Environmental Medicine)

Effect of an Integrative Medicine Course on Knowledge Base and Practice Patterns (Environmental Medicine)
http://integrativemedicine.arizona.edu/education/online_courses/enviro-med-study.html

Effect of an Integrative Medicine Course on Knowledge Base and Practice Patterns (Environmental Medicine)
The purpose of this study is to investigate the effectiveness of a brief, online course in improving health care provider knowledge, attitudes and skills and ability to apply clinical skills in routine patient care.

We expect approximately 50 people will take part in this study. If you decide to participate, you will be asked to:
Read and acknowledge a disclosure notification
Fill out a questionnaire before you take the course
Take an online course
Fill out a questionnaire right after you finish the course
Fill out a questionnaire 30 days after you complete the course
We estimate that it will take approximately 30 minutes to complete all questionnaires, 10 minutes each. There is no cost to you except for your time.

Industrial chemicals linked to attention problems in Massachusetts children

Industrial chemicals linked to attention problems in Massachusetts children
http://www.environmentalhealthnews.org/ehs/news/pcbs-and-attention-deficits-in-kids

"When Deidre Ramos moved with her infant son to the Parker Street section of New Bedford, Mass., little did she know that her new neighborhood was toxic. Today, a decade later, Ramos is worried about her two sons growing up in a community still contaminated by an old burn dump containing polychlorinated biphenyls. "What will be the long-term effects on my children?" asked Ramos. Now new research suggests that PCBs, which were first linked to learning problems in children more than two decades ago, may play a role in attention-deficit hyperactivity disorder, too. Boys in New Bedford who were exposed to higher levels of PCBs in the womb scored lower on focus and concentration tests, which indicates they are more likely to have attention problems related to ADHD, according to a newly published study. All of the children studied were born to mothers who lived near the contaminated harbor and dumpsites in this low-income community but their exposures were comparable to children's levels throughout the United States so the link to attention problems could exist in other communities, too."

Sunday, March 4, 2012

Global Indoor Health Network Position Statement: Common Toxins in our Homes, Schools and Workplaces

Global Indoor Health Network
Position Statement
Common Toxins in our Homes, Schools and Workplaces
http://globalindoorhealthnetwork.com/position_statement.html

NEWSLETTER: MCS America News - March 2012

MCSA NEWS
March 2012, Volume 7, Issue 3

 

Entire PDF Edition: http://mcs-america.org/March2012.pdf (View, Download, and Print)

 

Direct Links to Articles Inside This Issue:

 

MCS Guidelines for South Australian Hospitals

http://mcs-america.org/March2012pg12345678910.pdf

 

Weight Loss & Chemical Exposures

http://mcs-america.org/March2012pg1112.pdf  

 

Patient Support & Resources

http://mcs-america.org/March2012pg1314151617181920.pdf  

 

Community News

http://mcs-america.org/March2012pg21.pdf  

 

Featured Research Studies

http://mcs-america.org/March2012pg222324.pdf  

 

Multiple Chemical Sensitivities America

http://www.mcs-america.org

admin@mcs-america.org

 

Copyrighted © 2012 MCS America

 

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Saturday, March 3, 2012

Biological Factors that Underlie Individual Susceptibility to Environmental Stressors and Their Implications for Decision-Making

Biological Factors that Underlie Individual Susceptibility to Environmental Stressors and Their Implications for Decision-Making
http://nas-sites.org/emergingscience/workshops/individual-variability/

April 18-19, 2012

The National Academies
Keck Center
500 Fifth St. NW
Washington, DC 20001

The Emerging Science of BMAA: Do Cyanobacteria Contribute to Neurodegenerative Disease?

The Emerging Science of BMAA: Do Cyanobacteria Contribute to Neurodegenerative Disease?
 
Citation: Holtcamp W 2012. The Emerging Science of BMAA: Do Cyanobacteria Contribute to Neurodegenerative Disease? Environ Health Perspect 120:a110-a116. http://dx.doi.org/10.1289/ehp.120-a110
 
"In the late 1990s ethnobotanist Paul Alan Cox visited the indigenous Chamorro people of Guam, sleuthing for cancer cures in the lush rainforest. He soon stumbled upon troubling facts that would change the trajectory of his career, leading to major clues in understanding Lou Gehrig's disease (amyotrophic lateral sclerosis, or ALS) and possibly other neurodegenerative diseases. Since that time, major breakthroughs in the fields of neurobiology, epidemiology, and ecology have led to an increased interest in an unlikely hypothesis: that β-methylamino-ʟ-alanine (BMAA)—a cyanobacterial neurotoxin found in contaminated seafood and shellfish, drinking water supplies, and recreational waters—may be a major factor in these diseases."

Long-delayed diesel study published

Long-delayed diesel study published
http://www.iwatchnews.org/2012/03/02/8309/long-delayed-diesel-study-published

"A much-anticipated government study of more than 12,000 miners — whose publication was delayed by litigation from a group of mining companies — has found that exposure to diesel engine exhaust significantly increases the risk of lung cancer."

Long-delayed diesel study published

Long-delayed diesel study published
http://www.iwatchnews.org/2012/03/02/8309/long-delayed-diesel-study-published

A much-anticipated government study of more than 12,000 miners — whose publication was delayed by litigation from a group of mining companies — has found that exposure to diesel engine exhaust significantly increases the risk of lung cancer.

For the most heavily exposed miners, the risk of dying from lung cancer was three times higher than it was for those exposed to low doses. For non-smokers, the risk was seven times higher.

"[T]he findings suggest that the risks may extend to other workers exposed to diesel exhaust in the United States and abroad, and to people living in urban areas where diesel exhaust levels are elevated," Joseph Fraumeni Jr., director of the National Cancer Institute's Division of Cancer Epidemiology and Genetics, said in a press release Friday morning.

Two papers detailing the study's results were published in the Journal of the National Cancer Institute. One of them concluded that diesel-induced lung cancer "may represent a potential public health burden."

In an editorial in the journal, Lesley Rushton, an epidemiologist at Imperial College in London, wrote, "These results indicate that stringent occupational and particu­larly environmental standards for [diesel exhaust] should be set and compli­ance ensured to have an impact on health outcomes." The setting of stricter standards could have broad implications given the ubiquity of diesel engines in trucks, buses, ships, trains and construction equipment.

According to the NCI press release, the study is "the first … based on historical exposure to diesel exhaust to yield a statistically significant, positive increase in lung cancer risk with increasing diesel exposure after taking smoking and other potential lung cancer risk factors into account."

The Center for Public Integrity reported last month that publication of the $11.5 million study, conceived in 1992 by the NCI and the National Institute for Occupational Safety and Health (NIOSH), had been held up by litigation from the Mining Awareness Resource Group, an industry organization that first challenged the research effort in 1996.

In court filings, lawyers for the group alleged that the study was "inaccurate and faulty" and would "likely spawn public concerns, regulatory actions, and lawsuits." They received favorable rulings from a federal judge in Louisiana, who ordered the Department of Health and Human Services to give the industry group and the House Committee on Education and the Workforce 90 days to review study data and documents before any papers could be published in scientific journals.

Last August, the judge held HHS — parent of the NCI and NIOSH — in contempt after finding that the agency had withheld study materials from the industry group and the committee. HHS insisted it had done nothing wrong; it appealed the contempt order and proceeded with plans to publish despite stern letters sent to several journals last month by industry lawyer Henry Chajet. In the letters, Chajet warned the journals' editors that publication "could cause our clients serious harm" and violate court orders.

In an emailed statement to the Center Friday afternoon, Chajet wrote that the Mining Awareness Resource Group — MARG — "is concerned by the [NCI] press release and the reported study conclusions, which are under review by a distinguished group of independent scientific and medical experts. When the review is complete, the experts and MARG will release it to the impacted employees, HHS, the Congress and the public.

"In the interim, MARG expresses its disappointment that HHS refused to provide the data and documents underlying the study to the study participants and independent scientific experts, before issuing its press release, which refusal was in violation of Congressional directives and a 2001 Federal Court Order."

The second paper published Friday in the Journal of the National Cancer Institute noted that "future occupational and environmental exposure levels to [diesel exhaust] should be less than those encountered during the study" due to the increasing use of cleaner-burning diesel engines.

"However," the paper's authors wrote, "there will continue to be [a] legacy of older equipment in operation, the extent and duration of this varying across different countries depending on economic prosperity. Certainly, many workers around the world, in mining and other industries and jobs, continue to be exposed … at levels similar to those observed in this study."

Allen Schaeffer, executive director of the Diesel Technology Forum, a nonprofit organization in Maryland that represents diesel engine manufacturers, fuel refiners and makers of emission control technology, cautioned against reading too much into the study.

"This research is really retrospective," Schaeffer said. "It looks at equipment and fuel that in some cases might have been 50 years old. From a practical perspective, equipment and fuel from as much as half a century ago bears little if any resemblance to modern technology." Emissions from newer diesel engines are "near zero," he said, and there probably aren't many older, dirtier engines still in use in the United States.

But John Froines, a professor of toxicology at UCLA who chairs the Scientific Review Panel on Toxic Air Contaminants for the California Environmental Protection Agency, said the study is "extremely important" in that it reinforces earlier research suggesting diesel exhaust could cause cancer. California deemed the pollutant a carcinogen in 1998 and adopted strict regulations limiting emissions of diesel particulate matter.

The International Agency for Research on Cancer (IARC), part of the World Health Organization, considers diesel exhaust "probably carcinogenic to humans." But IARC is scheduled to revisit the science in June and could be swayed by the HHS miner study to list diesel as a known cancer-causing agent. That, in turn, could lead to new regulations and make it more difficult for industry to defend itself in lawsuits.

Thursday, March 1, 2012

CDC's National Report on Exposure to Environmental Chemicals: Updated Tables February 2012

CDC's National Report on Exposure to Environmental Chemicals: Updated Tables
February 2012CDC's Division of Laboratory Sciences released new data to the
Fourth National Report on Human Exposure to Environmental Chemicals, an
ongoing assessment of the U.S. population's exposure to environmental
chemicals using biomonitoring.
http://www.cdc.gov/exposurereport/

The Updated Tables, February 2012 provides nationally-representative
biomonitoring data from CDC's National Health and Nutrition Examination
Survey (NHANES) that has become available since the publication of the
Fourth Report in 2009.

The Updated Tables, February 2012 include 66 chemicals with updated tables
and 34 newly added chemicals since the release of the Fourth Report.

The Updated Tables, February 2012 and additional resources are now available
on CDC's National Exposure Report webpage.

Gut Check: Do Interactions between Environmental Chemicals and Intestinal Microbiota Affect Obesity and Diabetes?

Gut Check: Do Interactions between Environmental Chemicals and Intestinal Microbiota Affect Obesity and Diabetes?
http://ehp03.niehs.nih.gov/article/fetchArticle.action;jsessionid=41F2811765FD84D4F39BD0731AEBDF14?articleURI=info%3Adoi%2F10.1289%2Fehp.120-a123a
 
Citation: Holtcamp W 2012. Gut Check: Do Interactions between Environmental Chemicals and Intestinal Microbiota Affect Obesity and Diabetes? Environ Health Perspect 120:a123-a123. http://dx.doi.org/10.1289/ehp.120-a123a
Online: 01 March 2012

The microbiota that populate human intestinal tracts vary substantially from person to person, and mounting evidence suggests these interindividual variations in gut microbiota affect how a person metabolizes chemicals they may be exposed to. A review of the literature on this topic directed attention to a new hypothesis: that interactions between gut ecology and environmental chemicals contribute to obesity and diabetes [EHP 120(3):332–339; Snedeker and Hay]. No study has yet directly addressed that hypothesis, but this review comments on the strengths and weaknesses of studies linking environmental chemicals to obesity and diabetes and identifies gaps in the knowledge of how gut microbiota may affect the metabolism of these chemicals.

Studies reviewed by the authors found that differences in gut microbiota affected the toxicity of certain pharmaceuticals, including acetaminophen and the chemotherapy medication CPT-11. The authors propose that the same mechanisms may be at work with environmental chemicals. Enzymes produced by different gut microbe species can render ingested chemicals either more or less bioavailable, thereby affecting their toxicity. In reviewing evidence on 3 dozen suspected obesogenic and diabetogenic chemicals, the authors found that at least three-quarters of them may be metabolized by gut microbe enzymes in a way that affects their absorption, distribution, metabolism, and excretion.

Human studies have shown that obese and diabetic individuals have different gut microbiota compositions than lean and nondiabetic individuals. Likewise, bariatric surgery was associated with altered gut microbiota. The composition of a gut's microbiome may influence body weight by regulating fat storage, altering the ability of the intestines to extract energy from food, and affecting satiety by modulating the levels of hormones that regulate appetite.

The reviewers also found developmental links between gut microbiota and obesity. For instance, one study connected microbiome composition of children to their mother's weight, body mass index, and degree of weight gain during pregnancy, while another linked differences in infant microbiota composition to weight gain years later. The review highlights the importance of understanding how differences in gut microbiota might affect the fate of environmental chemicals in people and influence human health outcomes.

Stop the Biotech Bullies!


SUBSCRIBE & Read Past Issues | OCA Homepage |

Stop the Biotech Bullies!

If Monsanto's genetically engineered crops (corn, soybeans, canola, sugar beets, cotton) pollute an organic or non-GMO farmer's land, under corporate-corrupted U.S. patent law, Monsanto can sue these victimized farmers for so-called "theft of intellectual property." Since the mid-1990s, 144 farmers have been taken to court by the Biotech Bully of St. Louis. Monsanto has brought charges against more than 700 additional farmers who have settled out-of-court. These farmers had no intention of growing crops or saving seeds that contain Monsanto's controversial patented genes. Their crops have been contaminated by genetically engineered plants in neighboring fields or contamination in seed processing facilities.

In an attempt to protect these farmers, a case, Organic Seed Growers & Trade Association, et al. v. Monsanto, was filed in federal district court in Manhattan on March 29, 2011, on behalf of 83 family farmers, seed businesses and organic agricultural organizations, representing over 300,000 members.

On February 24, Judge Naomi Buchwald dismissed in the case.


Support Farmer Protection Legislation
 
Learn More & Donate to the Farmers' Appeal

 
 
 

Monsanto's Minions Attack OCA in California: Lies & Censorship at the Sacramento Bee

Last week, we reported that the Sacramento Bee had run an opinion piece by Dan Morain full of lies about the California ballot initiative campaign to label GMOs. We asked Bytes readers to set the record straight by reading our rebuttal and posting comments to the article on the Sacramento Bee website.

A flood of comments ensued, prompting the Bee to shut down the comment function. Then, on Saturday, Stuart Leavenworth, the editorial page editor, published a piece attacking the OCA that, like Morain's editorial, parrots Monsanto's talking points with no regard for the truth.

Again, we ask you to read Leavenworth's article and our rebuttal, then post comments defending freedom of speech and our right to know what's in our food.

Learn More & Take Action

 
 
 

Op-Ed of the Week: "America's Obesity and Diabetes Epidemic: Junk Food Kills"
by Ronnie Cummins

"The U.S. industrial food and farming system, dominated by fast food restaurants and processed, chemical-laden food, has precipitated a public health crisis. Although nutritionists recommend that consumers avoid eating unhealthy junk foods, every day 75 million Americans "supersize" themselves and damage their health by eating at McDonald's or other fast food restaurants. Forty percent of American meals are now purchased and consumed outside the home, typically consisting of high-calorie, low-nutrition items such as soft drinks, French fries, and low-grade meat, laced with fat, cheap sweeteners, pesticide residues, chemical additives, and salt. We have become a Fast Food Nation of bulging waistlines and high blood pressure."

Read More

 
 
 

Support the OCA and the OCF: Strike a Blow Against Biotech and Junk Food

OCA and our lobbying ally, the Organic Consumers Fund, put our money where our mouth is. With the 2012 California Ballot Initiative, and GMO labeling legislation gaining momentum in Vermont, Connecticut, Hawaii and other states, we have a real chance to hit Monsanto, corporate agribusiness, and the supermarket chains with the skull and crossbones they fear so much: mandatory labels on genetically engineered food. At the same time we are continuing our struggle to strengthen organic standards and significantly expand the market for organic food and products, especially those produced locally and regionally; while warning consumers about the dangers of junk food and chemical agriculture.

Donate to the Organic Consumers Association (tax-deductible)

Donate to the Organic Consumers Fund (non-tax-deductible, but necessary for our legislative efforts in California and other states)

 
 
 

Two More Months to Stop Dow's Agent Orange Frankencorn

The US Department of Agriculture has extended its comment period on Dow's request to commercialize corn which has been genetically engineered for increased resistance to the herbicide 2,4-D of Agent Orange infamy.

The comment deadline has been extended to April 27, 2012.

Learn More & Take Action

 

 
 
 

Little Bytes: Top Stories of the Week

Big Food Must Go: Why We Need to Radically Change the Way We Eat

'Natural': The Most Meaningless Word on Your Food Label?

Time for Environmental Funders to Stop Neglecting the Grassroots

Dr. Vandana Shiva: Occupy Our Food Supply!

Infiltration to Disrupt, Divide and Misdirect Is Widespread in Occupy

 
 
 

Message From Our Sponsors

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Wednesday, February 29, 2012

Transgenerational Actions of Environmental Compounds on Reproductive Disease and Identification of Epigenetic Biomarkers of Ancestral Exposures

Transgenerational Actions of Environmental Compounds on Reproductive Disease and Identification of Epigenetic Biomarkers of Ancestral Exposures
http://www.plosone.org/article/info:doi/10.1371/journal.pone.0031901

Mohan Manikkam#, Carlos Guerrero-Bosagna#, Rebecca Tracey, Md. M. Haque, Michael K. Skinner*
Center for Reproductive Biology, School of Biological Sciences, Washington State University, Pullman, Washington, United States of America

Abstract
Environmental factors during fetal development can induce a permanent epigenetic change in the germ line (sperm) that then transmits epigenetic transgenerational inheritance of adult-onset disease in the absence of any subsequent exposure. The epigenetic transgenerational actions of various environmental compounds and relevant mixtures were investigated with the use of a pesticide mixture (permethrin and insect repellant DEET), a plastic mixture (bisphenol A and phthalates), dioxin (TCDD) and a hydrocarbon mixture (jet fuel, JP8). After transient exposure of F0 gestating female rats during the period of embryonic gonadal sex determination, the subsequent F1–F3 generations were obtained in the absence of any environmental exposure. The effects on the F1, F2 and F3 generations pubertal onset and gonadal function were assessed. The plastics, dioxin and jet fuel were found to promote early-onset female puberty transgenerationally (F3 generation). Spermatogenic cell apoptosis was affected transgenerationally. Ovarian primordial follicle pool size was significantly decreased with all treatments transgenerationally. Differential DNA methylation of the F3 generation sperm promoter epigenome was examined. Differential DNA methylation regions (DMR) were identified in the sperm of all exposure lineage males and found to be consistent within a specific exposure lineage, but different between the exposures. Several genomic features of the DMR, such as low density CpG content, were identified. Exposure-specific epigenetic biomarkers were identified that may allow for the assessment of ancestral environmental exposures associated with adult onset disease.

Pollutants long gone, but disease carries on

Pollutants long gone, but disease carries on
 
"Certain chemicals cause epigenetic changes that foster illness in rats' offspring"

Metals from hip replacements present toxic risk for millions, investigation warns

Metals from hip replacements present toxic risk for millions, investigation warns
http://www.cbsnews.com/8301-504763_162-57387520-10391704/metals-from-hip-replacements-present-toxic-risk-for-millions-investigation-warns/

"(CBS News) Millions of people with metal on metal hip replacements may have been exposed to dangerously high levels of toxins that seeped into their bloodstreams, warned the researchers behind a British Medical Journal and BBC Newsnight investigation."

Environmental Obesogens Make Children and Adults Fat

Environmental Obesogens Make Children and Adults Fat
http://www.i-sis.org.uk/Environmental_Obesogens_make_children_and_adults_fat.php

Tuesday, February 28, 2012

Early diphtheria-tetanus-pertussis vaccination associated with higher female mortality and no difference in male mortality in a cohort of low birthweight children: an observational study within a randomised trial

Early diphtheria-tetanus-pertussis vaccination associated with higher female mortality and no difference in male mortality in a cohort of low birthweight children: an observational study within a randomised trial
http://adc.bmj.com/content/early/2012/02/13/archdischild-2011-300646.short?g=w_adc_ahead_tab

Arch Dis Child doi:10.1136/archdischild-2011-300646

Background: Studies from low-income countries have suggested that diphtheria-tetanus-pertussis (DTP) vaccine provided after Bacille Calmette-Guerin (BCG) vaccination may have a negative effect on female survival. The authors examined the effect of DTP in a cohort of low birthweight (LBW) infants.

Methods: 2320 LBW newborns were visited at 2, 6 and 12 months of age to assess nutritional and vaccination status. The authors examined survival until the 6-month visit for children who were DTP vaccinated and DTP unvaccinated at the 2-month visit.

Results: Two-thirds of the children had received DTP at 2 months and 50 deaths occurred between the 2-month and 6-month visits. DTP vaccinated children had a better anthropometric status for all indices than DTP unvaccinated children. Small mid-upper arm circumference (MUAC) was the strongest predictor of mortality. The death rate ratio (DRR) for DTP vaccinated versus DTP unvaccinated children differed significantly for girls (DRR 2.45; 95% CI 0.93 to 6.45) and boys (DRR 0.53; 95% CI 0.23 to 1.20) (p=0.018, homogeneity test). Adjusting for MUAC, the overall effect for DTP vaccinated children was 2.62 (95% CI 1.34 to 5.09); DRR was 5.68 (95% CI 1.83 to 17.7) for girls and 1.29 (95% CI 0.56 to 2.97) for boys (p=0.023, homogeneity test). While anthropometric indices were a strong predictor of mortality among boys, there was little or no association for girls.

Conclusion: Surprisingly, even though the children with the best nutritional status were vaccinated early, early DTP vaccination was associated with increased mortality for girls.

Toxic chemical found under O.C. building

Toxic chemical found under O.C. building
http://www.latimes.com/news/local/la-me-toxic-building-20120228,0,6688148.story

"Levels of tetrachloroethylene exceeding safety standards are found beneath offices used by Orange County agencies, including the Sheriff's Department and Social Services Agency. The building is already the subject of two lawsuits."

Low Levels of Omega-3 Fatty Acids May Cause Memory Problems

Low Levels of Omega-3 Fatty Acids May Cause Memory Problems
http://www.sciencedaily.com/releases/2012/02/120227162549.htm

"A diet lacking in omega-3 fatty acids, nutrients commonly found in fish, may cause your brain to age faster and lose some of its memory and thinking abilities, according to a study published in the February 28, 2012, print issue of Neurology®, the medical journal of the American Academy of Neurology. Omega-3 fatty acids include the nutrients called docosahexaenoic acid (DHA) and eicosapentaenoic acid (EPA)."

Monday, February 27, 2012

Infant mortality rates regressed against number of vaccine doses routinely given: is there a biochemical or synergistic toxicity?

Infant mortality rates regressed against number of vaccine doses routinely given: is there a biochemical or synergistic toxicity?
http://het.sagepub.com/content/30/9/1420.long
 
Miller NZ, Goldman GS.  Hum Exp Toxicol. 2011 Sep;30(9):1420-8.

"The infant mortality rate (IMR) is one of the most important indicators of the socio-economic well-being and public health conditions of a country. The US childhood immunization schedule specifies 26 vaccine doses for infants aged less than 1 year--the most in the world--yet 33 nations have lower IMRs. Using linear regression, the immunization schedules of these 34 nations were examined and a correlation coefficient of r = 0.70 (p < 0.0001) was found between IMRs and the number of vaccine doses routinely given to infants. Nations were also grouped into five different vaccine dose ranges: 12-14, 15-17, 18-20, 21-23, and 24-26. The mean IMRs of all nations within each group were then calculated. Linear regression analysis of unweighted mean IMRs showed a high statistically significant correlation between increasing number of vaccine doses and increasing infant mortality rates, with r = 0.992 (p = 0.0009). Using the Tukey-Kramer test, statistically significant differences in mean IMRs were found between nations giving 12-14 vaccine doses and those giving 21-23, and 24-26 doses. A closer inspection of correlations between vaccine doses, biochemical or synergistic toxicity, and IMRs is essential."



















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